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What are complications of acute glomerulonephritis

Sep 11, 2017

This article introduction: acute glomerulonephritis is one of the nephritis, has a great influence on kidney, and easy to cause complications, here are together and see what are the complications of acute glomerulonephritis.

The main complications in acute stage Serious complications are mainly in the acute phase of the cycle of severe congestion state, hypertensive encephalopathy, and acute renal failure.As in recent years, prevention and control work to strengthen its incidence and case fatality rate has decreased obviously.

1. The circulation pump: for water sodium retention, dragon's blood volume, and pulmonary edema.The incidence of various reports, related to the illness weight, the treatment situation.In 50 ~ 60 s reported in 24% ~ 27% of hospitalized children with acute glomerulonephritis seen such complications, in recent years, the report had fallen to 2.4%.Develops in 1 ~ 2 weeks after the onset of the acute nephritis.Clinical expression is short of breath, cannot lay down, chest tightness, cough, lung wet rale, liver tenderness, gallop about heart failure symptoms, such as is caused by a blood volume expansion, and is different from real myocardial pump exhaust.At heart a decrease more often not, cycle time is normal, arteriovenous difference of oxygen partial pressure did not see more, and digitalis class cardiotonic effect not beautiful, and the application of diuretics can often make their ease.A handful of intensive development to the real heart failure, rapid emergence within a few hours to 1 ~ 2 days life-threatening pulmonary edema.

2. Hypertensive encephalopathy: refers to the blood pressure (especially the diastolic blood pressure increased sharply, in terms of central nervous system symptoms.General children than adults.Disease is often thought to be on the basis of systemic hypertension, small resistance in the brain vasospasm lead to lack of brain edema and causes;But some people think that is a dramatic increase in blood pressure, cerebral original have automatic diastolic regulating function of out of control, cerebrovascular highly congested, cerebral edema and acute nephritis in addition to the water sodium retention may also play a role in the pathogenesis.Develops in the early acute nephritis course, generally more urgent, characterized by severe headache, nausea and vomiting frequently, then visual impairment, temporary amaurosis, giddiness, diplopia, and drowsiness or irritability, if not treated, convulsions, coma, a temporary hemiplegia, aphasia, serious when cerebral hernia.Nervous system does not have a limit signs, more shallow reflection and tendon reflex can be reduced or disappeared, ankle clonus positive, sometimes also can appear the pathological reflex, severe cases can have the signs and symptoms of cerebral hernia.Common retinal fundus examination small artery spasm, sometimes visible papilledema.Cerebrospinal fluid and clear, the pressure and protein normal or increased slightly.Such as blood pressure more than 18.7/12.0 kPa (18.7/12.0 MMHG), and associated with visual impairment, convulsions and coma is one of the three items can be diagnosed.

Acute nephritis in children with acute renal failure: a part in the acute phase with varying degrees of nitrogen qualitative hematic disease, but is only a handful of progression to acute renal failure.Complications is still lack of effective prevention measures, has become the leading cause of death in acute nephritis.Clinical manifestations of oliguria or anuria, blood urea nitrogen, serum creatinine increased, high potassium and metabolic acidosis.Oliguria or anuria for 3 ~ 5 or more than 1 week, then increase gradually, symptoms, function of urine.

 

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